"Virus Activation" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The mechanism by which latent viruses, such as genetically transmitted tumor viruses (PROVIRUSES) or PROPHAGES of lysogenic bacteria, are induced to replicate and then released as infectious viruses. It may be effected by various endogenous and exogenous stimuli, including B-cell LIPOPOLYSACCHARIDES, glucocorticoid hormones, halogenated pyrimidines, IONIZING RADIATION, ultraviolet light, and superinfecting viruses.
| Descriptor ID |
D014775
|
| MeSH Number(s) |
G06.920.925.940
|
| Concept/Terms |
Virus Activation- Virus Activation
- Activation, Virus
- Activations, Virus
- Virus Activations
- Viral Activation
- Activation, Viral
- Activations, Viral
- Viral Activations
- Virus Induction
- Induction, Virus
- Inductions, Virus
- Virus Inductions
Prophage Induction- Prophage Induction
- Induction, Prophage
- Inductions, Prophage
- Prophage Inductions
Prophage Excision- Prophage Excision
- Excision, Prophage
- Excisions, Prophage
- Prophage Excisions
|
Below are MeSH descriptors whose meaning is more general than "Virus Activation".
Below are MeSH descriptors whose meaning is more specific than "Virus Activation".
This graph shows the total number of publications written about "Virus Activation" by people in this website by year, and whether "Virus Activation" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2001 | 1 | 1 | 2 |
| 2002 | 1 | 0 | 1 |
| 2004 | 0 | 1 | 1 |
| 2005 | 0 | 1 | 1 |
| 2006 | 0 | 1 | 1 |
| 2007 | 0 | 1 | 1 |
| 2008 | 1 | 0 | 1 |
| 2009 | 1 | 0 | 1 |
| 2010 | 0 | 1 | 1 |
| 2017 | 0 | 1 | 1 |
| 2018 | 1 | 0 | 1 |
| 2020 | 1 | 0 | 1 |
| 2021 | 1 | 0 | 1 |
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Below are the most recent publications written about "Virus Activation" by people in Profiles.
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Herpes Simplex Virus 1 MicroRNA miR-H8 Is Dispensable for Latency and Reactivation In Vivo. J Virol. 2021 01 28; 95(4).
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Deletion of Herpes Simplex Virus 1 MicroRNAs miR-H1 and miR-H6 Impairs Reactivation. J Virol. 2020 07 16; 94(15).
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Herpes Simplex Virus 1 Mutant with Point Mutations in UL39 Is Impaired for Acute Viral Replication in Mice, Establishment of Latency, and Explant-Induced Reactivation. J Virol. 2018 04 01; 92(7).
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Atypical herpes zoster presentation in a healthy vaccinated pediatric patient. Cutis. 2017 Nov; 100(5):303-304.
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Pronounced virus-dependent activation drives exhaustion but sustains IFN-? transcript levels. J Immunol. 2010 Sep 15; 185(6):3643-51.
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RIG-I-mediated activation of p38 MAPK is essential for viral induction of interferon and activation of dendritic cells: dependence on TRAF2 and TAK1. J Biol Chem. 2009 Apr 17; 284(16):10774-82.
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Delivery of Interferon-gamma by an adenovirus vector blocks herpes simplex virus Type 1 reactivation in vitro and in vivo independent of RNase L and double-stranded RNA-dependent protein kinase pathways. J Neuroimmunol. 2009 Jan 03; 206(1-2):39-43.
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The Rta/Orf50 transactivator proteins of the gamma-herpesviridae. Curr Top Microbiol Immunol. 2007; 312:71-100.
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Herpes simplex virus and the chemokines that mediate the inflammation. Curr Top Microbiol Immunol. 2006; 303:47-65.
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The Herpesvirus saimiri replication and transcription activator acts synergistically with CCAAT enhancer binding protein alpha to activate the DNA polymerase promoter. J Virol. 2005 Nov; 79(21):13548-60.