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Induction of G protein-coupled receptor kinases 2 and 3 contributes to the cross-talk between mu and ORL1 receptors following prolonged agonist exposure.
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Induction of G protein-coupled receptor kinases 2 and 3 contributes to the cross-talk between mu and ORL1 receptors following prolonged agonist exposure.
Induction of G protein-coupled receptor kinases 2 and 3 contributes to the cross-talk between mu and ORL1 receptors following prolonged agonist exposure. Neuropharmacology. 2002 Nov; 43(6):979-90.
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PubMed
subject areas
Analysis of Variance
Analysis of Variance
beta-Adrenergic Receptor Kinases
beta-Adrenergic Receptor Kinases
Cell Membrane
Cell Membrane
Cyclic AMP
Cyclic AMP
Cyclic AMP-Dependent Protein Kinases
Cyclic AMP-Dependent Protein Kinases
Dose-Response Relationship, Drug
Dose-Response Relationship, Drug
Drug Interactions
Drug Interactions
Enkephalin, Ala(2)-MePhe(4)-Gly(5)-
Enkephalin, Ala(2)-MePhe(4)-Gly(5)-
Enzyme Inhibitors
Enzyme Inhibitors
Flavonoids
Flavonoids
G-Protein-Coupled Receptor Kinase 3
G-Protein-Coupled Receptor Kinase 3
Humans
Humans
Mitogen-Activated Protein Kinases
Mitogen-Activated Protein Kinases
Morphine
Morphine
Neuroblastoma
Neuroblastoma
Oligodeoxyribonucleotides, Antisense
Oligodeoxyribonucleotides, Antisense
Opioid Peptides
Opioid Peptides
Receptor Cross-Talk
Receptor Cross-Talk
Receptors, Opioid
Receptors, Opioid
Receptors, Opioid, mu
Receptors, Opioid, mu
Transcription Factors
Transcription Factors
Tumor Cells, Cultured
Tumor Cells, Cultured
authors with profiles
Kelly M Standifer