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CCAAT/enhancing binding protein beta deletion in mice attenuates inflammation, endoplasmic reticulum stress, and lipid accumulation in diet-induced nonalcoholic steatohepatitis.
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CCAAT/enhancing binding protein beta deletion in mice attenuates inflammation, endoplasmic reticulum stress, and lipid accumulation in diet-induced nonalcoholic steatohepatitis.
CCAAT/enhancing binding protein beta deletion in mice attenuates inflammation, endoplasmic reticulum stress, and lipid accumulation in diet-induced nonalcoholic steatohepatitis. Hepatology. 2007 May; 45(5):1108-17.
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PubMed
subject areas
Acute-Phase Reaction
Acute-Phase Reaction
Animals
Animals
Body Weight
Body Weight
CCAAT-Enhancer-Binding Protein-beta
CCAAT-Enhancer-Binding Protein-beta
Choline Deficiency
Choline Deficiency
Cytochrome P-450 CYP2E1
Cytochrome P-450 CYP2E1
Diet
Diet
Disease Models, Animal
Disease Models, Animal
eIF-2 Kinase
eIF-2 Kinase
Endoplasmic Reticulum
Endoplasmic Reticulum
Eukaryotic Initiation Factor-2
Eukaryotic Initiation Factor-2
Fatty Liver
Fatty Liver
Inflammation
Inflammation
JNK Mitogen-Activated Protein Kinases
JNK Mitogen-Activated Protein Kinases
Lipase
Lipase
Liver
Liver
Male
Male
Methionine
Methionine
Mice
Mice
Mice, Inbred C57BL
Mice, Inbred C57BL
NF-kappa B
NF-kappa B
Transcription Factor CHOP
Transcription Factor CHOP
Tumor Necrosis Factor-alpha
Tumor Necrosis Factor-alpha
authors with profiles
Jacob Friedman
Rachel C Janssen