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Oxidative stress alters base excision repair pathway and increases apoptotic response in apurinic/apyrimidinic endonuclease 1/redox factor-1 haploinsufficient mice.
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Oxidative stress alters base excision repair pathway and increases apoptotic response in apurinic/apyrimidinic endonuclease 1/redox factor-1 haploinsufficient mice.
Oxidative stress alters base excision repair pathway and increases apoptotic response in apurinic/apyrimidinic endonuclease 1/redox factor-1 haploinsufficient mice. Free Radic Biol Med. 2009 Jun 01; 46(11):1488-99.
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PubMed
subject areas
Animals
Animals
Apoptosis
Apoptosis
Carrier Proteins
Carrier Proteins
Caspases
Caspases
DNA Damage
DNA Damage
DNA Glycosylases
DNA Glycosylases
DNA Repair
DNA Repair
DNA-(Apurinic or Apyrimidinic Site) Lyase
DNA-(Apurinic or Apyrimidinic Site) Lyase
Enzyme Activation
Enzyme Activation
Intracellular Signaling Peptides and Proteins
Intracellular Signaling Peptides and Proteins
Liver
Liver
Mice
Mice
NF-kappa B
NF-kappa B
Nitroparaffins
Nitroparaffins
Oxidative Stress
Oxidative Stress
Propane
Propane
Protein Binding
Protein Binding
Protein Stability
Protein Stability
Tumor Suppressor Protein p53
Tumor Suppressor Protein p53
Uracil-DNA Glycosidase
Uracil-DNA Glycosidase
authors with profiles
Archana Unnikrishnan