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Doublecortin-like kinase 1 promotes hepatocyte clonogenicity and oncogenic programming via non-canonical ß-catenin-dependent mechanism.
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Doublecortin-like kinase 1 promotes hepatocyte clonogenicity and oncogenic programming via non-canonical ß-catenin-dependent mechanism.
Doublecortin-like kinase 1 promotes hepatocyte clonogenicity and oncogenic programming via non-canonical ß-catenin-dependent mechanism. Sci Rep. 2020 06 29; 10(1):10578.
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subject areas
alpha-Fetoproteins
alpha-Fetoproteins
Animals
Animals
beta Catenin
beta Catenin
Carcinogenesis
Carcinogenesis
Carcinoma, Hepatocellular
Carcinoma, Hepatocellular
Cell Line, Tumor
Cell Line, Tumor
Glycogen Synthase Kinase 3 beta
Glycogen Synthase Kinase 3 beta
Hep G2 Cells
Hep G2 Cells
Hepatocytes
Hepatocytes
Heterografts
Heterografts
Humans
Humans
Intracellular Signaling Peptides and Proteins
Intracellular Signaling Peptides and Proteins
Liver Cirrhosis
Liver Cirrhosis
Liver Neoplasms
Liver Neoplasms
Mice
Mice
Mice, Inbred C57BL
Mice, Inbred C57BL
Neoplastic Stem Cells
Neoplastic Stem Cells
SOX9 Transcription Factor
SOX9 Transcription Factor
Spheroids, Cellular
Spheroids, Cellular
authors with profiles
Naushad Ali
Courtney Wayne Houchen
Dongfeng Qu
Javid Fazili
Mark M Huycke
Michael Stuart Bronze
Min Li
Randal Joseph May