Orphanin FQ/nociceptin-mediated desensitization of opioid receptor-like 1 receptor and mu opioid receptors involves protein kinase C: a molecular mechanism for heterologous cross-talk.

Orphanin FQ/nociceptin-mediated desensitization of opioid receptor-like 1 receptor and mu opioid receptors involves protein kinase C: a molecular mechanism for heterologous cross-talk. J Pharmacol Exp Ther. 2002 Aug; 302(2):502-9.

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subject areas

Alkaloids
Alkaloids
Benzophenanthridines
Benzophenanthridines
beta-Adrenergic Receptor Kinases
beta-Adrenergic Receptor Kinases
Cell Membrane
Cell Membrane
Cyclic AMP
Cyclic AMP
Cyclic AMP-Dependent Protein Kinases
Cyclic AMP-Dependent Protein Kinases
Enkephalin, Ala(2)-MePhe(4)-Gly(5)-
Enkephalin, Ala(2)-MePhe(4)-Gly(5)-
Enzyme Inhibitors
Enzyme Inhibitors
G-Protein-Coupled Receptor Kinase 3
G-Protein-Coupled Receptor Kinase 3
Humans
Humans
Neuroblastoma
Neuroblastoma
Oligodeoxyribonucleotides, Antisense
Oligodeoxyribonucleotides, Antisense
Opioid Peptides
Opioid Peptides
Phenanthridines
Phenanthridines
Protein Kinase C
Protein Kinase C
Receptor Cross-Talk
Receptor Cross-Talk
Receptors, Opioid
Receptors, Opioid
Receptors, Opioid, mu
Receptors, Opioid, mu
Tetradecanoylphorbol Acetate
Tetradecanoylphorbol Acetate
Tumor Cells, Cultured
Tumor Cells, Cultured

authors with profiles

Kelly M Standifer