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Increased cAMP in monocytes augments Notch signaling mechanisms by elevating RBP-J and transducin-like enhancer of Split (TLE).
The design, synthesis and biological evaluation of conformationally restricted 4-substituted-2,6-dimethylfuro[2,3-d]pyrimidines as multi-targeted receptor tyrosine kinase and microtubule inhibitors as potential antitumor agents.
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Increased cAMP in monocytes augments Notch signaling mechanisms by elevating RBP-J and transducin-like enhancer of Split (TLE).
Increased cAMP in monocytes augments Notch signaling mechanisms by elevating RBP-J and transducin-like enhancer of Split (TLE). J Biol Chem. 2013 Jul 26; 288(30):21526-36.
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PubMed
subject areas
Animals
Antigens, Bacterial
Bacterial Toxins
Basic Helix-Loop-Helix Transcription Factors
Bucladesine
CCAAT-Enhancer-Binding Protein-beta
Cell Line
Cells, Cultured
Co-Repressor Proteins
Cyclic AMP
Gene Expression
HEK293 Cells
Homeodomain Proteins
Humans
Immunoblotting
Immunoglobulin J Recombination Signal Sequence-Binding Protein
Lipopolysaccharides
Luciferases
Macrophages
Mice
Mice, Inbred C57BL
Monocytes
Protein Binding
Receptor, Notch1
Repressor Proteins
Reverse Transcriptase Polymerase Chain Reaction
Signal Transduction
Transcription Factor HES-1
authors with profiles
Jimmy D. Ballard
Jason L Larabee